Lol, I am not confused in the slightest....
The problem however is the person that thinks they are the smartest person in the room - far too often isn't - even though they think and pontificate like they are.
Find a mirror, go stand in front of it look at your reflection and read the above statement.
From a johns hopkins white paper....
SARS-CoV-2 Genetics Updated April 16, 2020 Key Findings for Public Health •
The outbreak was initiated from either a single introduction into humans or very few animal-to-human transmission events. •
SARS-CoV-1 and SARS-CoV-2 use the same cellular receptor, ACE2, which could be used as a starting point for creating therapeutics for SARS-CoV-2. Background Coronaviruses, including the pneumonia-causing novel coronavirus currently known as SARS-CoV-2, are enveloped, nonsegmented, positive-sense RNA viruses. Coronavirus genomes have some of the largest genomes among RNA viruses, with approximately 25-32 kilobases.1 The typical CoV genome includes a 5’-cap, 5’-untranslated region (UTR), open reading frames, a 3’-UTR, and 3’-poly(A) tail. The first two thirds of the genome typically codes for nonstructural proteins from 2 open reading frames that form the replicase complex. The last third of the genome encodes primarily structural proteins.2 There are 4 conserved structural proteins across CoVs: the spike (S) protein, membrane (M) protein, envelope (E) protein, and nucleocapsid (N) protein.1 The S protein is responsible for binding to host cell receptors and viral entry to host cells. The M, E, and N proteins are part of the nucleocapsid of viral particles. SARS-CoV-2 Naming In a paper published early in the pandemic,3 viral sequences collected from the earliest patients were assessed and compared to known viral sequences. Sequence analysis of 11 samples found that SARS-CoV-2 is in the same species as SARS-CoV; the 2 viruses are 94.6% similar in amino acid sequence (80% nucleotide sequence similarity) across the genome.3 However, other studies from early in the outbreak do not consider the viruses to be the same species, as they differ by more than 10% in the replicase genes.4 In February, the Coronavirus Study Group (CSG) of the International Committee on Taxonomy of Viruses officially named the novel coronavirus SARS-CoV-2. The CSG analyzed viral genomes from several patients and assessed phylogenetic (evolutionary) relationships between the new virus and known coronaviruses. The committee found that the genome of viruses isolated from patients was similar enough to SARS genomes to be considered a variant of SARS, not an entirely novel virus. While the clinical presentation, epidemiologic patterns, and host range of SARS-CoV-2 may differ from the original SARS-CoV, it is the genetic similarity between the 2 viruses that is used to conclude they are the same species. For this reason, the CSG has named SARS-CoV and SARS-CoV-2 as variants of the species known as Severe acute respiratory syndrome– related coronaviruses. The name SARS-CoV-2 is distinct from the name of the disease, which the WHO has officially designated COVID-19.
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Given the fact that they are so closely related and share quite a lot, while genetically they are related they are different to a degree.
Given that HC and C, were considered for sars-cov, it is not unreasonable to think you couldn't apply it to sars-cov2.
Additionally, usage of it as a last resort is not the place to use it, some have determined. It has better results when used earlier...
By then clotting is tje major cause of organ failure, or so has been reported.
Stop being intentionally obtuse..... and a dolt.
I work with young folks like you daily, and they far often than not make asses of themselves. No matter how much paper you hang or letters you place after your name, it cannot give you common sense.
Now go find some blocks and play, adults are speaking